Research

BPC-157 and Tissue Repair: FAK/Paxillin Signaling and Nitric Oxide Pathway Activation

Mechanistic analysis of BPC-157 activity in tissue repair models: FAK phosphorylation at Tyr397, paxillin recruitment, Rac1/Cdc42 activation, VEGFR2 upregulation, and NO production in HUVEC. Quantitative data from tendon, muscle, and vascular tissue models.